 Posted: Sun 0:40, 13 Mar 2011 Post subject: Color Doppler ultrasound guidance for percutaneous |
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| Color Doppler ultrasound guidance for percutaneous nephrolithotomy lithotomy clinical value of
, Delay treatment, in clinical work, the experimental treatment for the differential diagnosis of MDS and MA, has practical value. 1 INFORMATION 1.1 General information from 2001 to 2008 patients in our hospital MDS18 cases. 1O cases of male and 8 female, aged 21 to 48 years, mean 42 years; MA group,p90x workout schedule, 26 patients aged 23 to 52 with a mean of 46 years. Diagnosis of blood diseases are in line with the appropriate diagnostic criteria ... 1, excluding the poor from the free solution,tory burch reva, PNH and other hemolytic diseases. 1.2 in both groups of oral folic acid treatment l0mg3 times / d,belstaff italia, vitamin Bl2100 im 1 / d. Experimental therapy for 1 month. 1.3 The dynamic observation of blood laboratory tests, serum lactate dehydrogenase (LDH), folic acid (FA), vitamin BI2 'serum total bilirubin (113 s). 1.4 ± s statistical data that was used to compare between the two groups t test. 2 Results Before treatment,timberland shoes, MDS,tory burch outlet, MA serum LDH level, rrBI 【J levels were significantly higher than normal. MA group 1 week after treatment and 1 month in serum LDH, TBm reduce and gradually return to normal, compared with before treatment was significant (P <0.O1), MDS group for 1 week and 1 month in serum LDH, TBIL compared with before treatment There was no significant difference. Table 1MA before and after treatment with the MDS LDH, TBIL, lib level of comparison (teeth ± 5) 3 discussions MDS are a group of clonal hematopoietic stem cell disease, because there is dysplasia, immature red blood cell hemolysis, shortened life expectancy, causes anemia, serum LDH, 1BIIJ levels were significantly increased. MA is due to folic acid, vitamin Bl2 lack of DNA induced blast cells generated in bone marrow disorder, red blood cells cause bone marrow megaloblastic change in situ hemolysis, increased destruction of red blood cells causes anemia, LDH, TBIL increased. MDS with MA were similar to early clinical misdiagnosis in the early dysplasia is not no typical MDS, morphology alone is difficult to identify with the MA, we retrospectively MA MDS and experimental changes in blood before and after treatment, LDH, TBIL News changes prompted by the folic acid and vitamin B MA】 2 treatment LDH, TBIL down and return to normal, Fib gradually increased and returned to normal, indicating that folic acid and vitamin B12 after treatment of cells mature MA obstacles to improve, in situ hemolysis decreased red blood cell destruction Jianshao . The MDS group LDH, TBIL continued high level, Hb no improvement, indicating that folic acid, vitamin Bl2 does not improve the MDS, dysplasia, both experimental treatment can be used as identification. I work in the usual clinical routine blood test for unknown reasons was pancytopenia who dysplasia is not obvious, making it difficult to conclude, the experimental treatment can be observed dynamically in January, to prevent misdiagnosis. |
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